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In the UC model, hydrogen sulfide, which shows increased levels in the intestinal tract with inflammation, stimulates cystine production and increases glutathione levels 27
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GLP-1 RAs link to gallbladder/biliary diseases without increasing pancreatitis risk
During the past decade, metabolic reprogramming, particularly aberrant aerobic glycolysis in fibroblasts and macrophages, has been confirmed to play a significant role in the pathogenesis of pulmonary fibrosis [11]
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