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Although extracellular vesicles, oxidized phospholipids, secreted proteins, and lipid-derived electrophilic metabolites all represent plausible candidates, it remains unclear which mediators possess sufficient stability and diffusivity to support propagation across anatomically separated yet functionally connected cells

A key repressor of this signaling, phosphatase and tensin homolog deleted on chromosome 10 (PTEN), dephosphorylates PIP 3 to PIP 2 regulating the activity of PI3K/AKT 66