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Glutathione is the anchor for brightening: the bodys master antioxidant inhibits tyrosinase, the enzyme that drives melanin production, which is the mechanism behind its use as a brightening and antioxidant support, not a disease treatment

MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)

We hypothesized that the hepatic metabolome would be similarly characterized by increased alternative energy pathway utilization, altered redox balance and glutathione depletion, and reduced levels of ATP

doi:10.1007/7081_2015_176