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Lin et al (166) demonstrated that biomimetic high-density lipoprotein nanoparticles could disrupt intracellular cholesterol homeostasis by targeting the scavenger receptor class B type 1 receptor, which was upregulated in AML cells
Values are reported as mean SEM
By activating two distinct but complementary pathways, the combination triggers a much more significant and controlled GH release in lab models than either peptide could ever manage on its own
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