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Importantly, we found that addition of FXR-antagonistic BAs, including TMCA, HDCA or UDCA, could antagonize the effect of CDCA, a potent FXR agonist, on the mRNA expression of Shp and GLP-1 secretion in NCI-H716 cells (Fig
Always verify total mg and concentration on the supplier's COA before drawing a dose
We reasoned that the enhanced response in knockout islets was attributed to a compensatory enhancement in GLP-1R signaling that has been previously described in various GIPR knockout models 10,21,22
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