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Supplementing with reduced glutathione supports this critical bottleneck, ensuring toxins can be safely packaged and removed
Core molecular mechanisms encompass the targeted modification of inflammatory signaling molecules [408], oxidative alteration of pathogen entry-related proteins [204], and direct damage to host lipids/DNA [409]
(34) shows that HK2 presents resistance to cisplatin in the ovarian cancer cell, promotes the resistance by enhancing cisplatin-induced autophagy, and boosts the cisplatin-induced autophagy by activating the ERK1/2 pathway
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