To further assess the in vivo antioxidant activity of the fraction (F3) which had a high DPPH radical scavenging rate, 60 male mice were randomly divided into 6 groups (normal, positive control, model group, and low-, medium-, and high-dose group), and immunosuppression was induced via intraperitoneal cyclophosphamide injection
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Conclusions In conclusion, UA can exert neuroprotective effects on neurodegenerative diseases by inhibiting oxidative stress and neuroinflammation
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