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An excess of Fe 2+ leads to the production of ROS through the Fenton reaction, which induces cell damage or death and potentially impairs organ function eventually

The four primary mechanisms are: signaling the hypothalamus to suppress hunger, slowing gastric emptying so fullness lasts longer, stimulating insulin release after meals to prevent blood sugar spikes, and suppressing glucagon to reduce glucose release from the liver

Notably, neutrophil death is often accompanied by the release of neutrophil extracellular traps (NETs), which activate Toll-like receptors and induce ROS production