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Individuals with a history of pulmonary, neurologic, hepatic, rheumatic, hematologic, or renal chronic disease, who were treated with immunosuppressive therapy in the 6 months prior to screening, or who used medication or supplements that may have confounded the study results were also excluded

Exogenous IGF-1 LR3 can suppress endogenous GH secretion through this feedback, potentially reducing pituitary output during the administration period [1]

Elevated JMJD3 expression has been consistently observed in human lung tumors and is particularly enriched in Ras-activated lung cancer cells

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