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The alkyl radical in the presence of oxygen is converted to peroxyl radicals which facilitate the long-term damage by continually regenerating themselves through hydrogen abstraction from the nearby biomolecules and forming the hydroperoxides as the byproduct (Fig
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Thus, we measured TrB1-3 distribution in B cell-specific Gclc -deficient mice and found that TrB1 were increased in Gclc -deficient mice compared to controls (Supplementary Fig
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