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Tendon injuries initially suffer from: Poor blood supply Limited vascularity Slow nutrient delivery In rodent models, BPC-157 appears to: Upregulate VEGFR2 signalling Increase capillary density Improve microvascular perfusion Mechanistically: BPC-157 VEGFR2 activation PI3K/Akt pathway eNOS phosphorylation Increased nitric oxide Improved angiogenesis More vascularity = better nutrient and oxygen delivery to fibroblasts
(Nature, 1980) reported facilitated copper uptake into cultured cells, Simon et al
GHK-Cu is a compound which has a long history of safe usage in skin care and which has been studied extensively in animal experiments, and in cell and tissue culture studies
PubMed Wang, Y., Feng, Z., & Yu, L
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