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LZ was supported by NIH grants (DC008983, MH116990, and DC020887)
JN: Conceptualization, Funding acquisition, Methodology, Resources, Supervision, Validation, Visualization, Writing review & editing
revealed that in an ISO-mediated model of myocardial infarction, direct overexpression of the Nrf2 gene enhanced GSH levels in cardiomyocytes and guarded cardiomyocytes against oxidative damage and lessened the occurrence of ferroptosis [202]
Under such conditions it is essential to provide the body with the key nutrients involved in GSH synthesis in order to sustain functionally appropriate levels of GSH
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