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After treatment with CoCl, BPC157-treated HUVECs failed to form tube-like structures (Supplementary Figure S3B), which is consistent with our analysis that the redox state is a key determinant of BACH1s functional direction
Recent research shows it alleviates inflammation in conditions like silicosis and ulcerative colitis by targeting pathways like SIRT1/STAT3, promoting tissue repair and reducing oxidative stress without systemic toxicity (10)
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