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The combination of higher bioavailability, faster onset, and ability to inject near the injury site provides advantages that oral administration cannot match
Autophagy exhibits dual roles in LPS cardiotoxicity: cytoprotective autophagy via the TNF- /p38MAPK/NOS axis, where oxidative stress triggers LC3-II conversion to limit LPS-mediated ROS overproduction (Yuan et al., 2009)
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How might this neuroimmune approach transform treatment strategies for other autoimmune conditions
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