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In ARDS, an imbalance between these pathways contributes to exaggerated immune responses, tissue damage, and disease progression ( 2.1 Ferroptosis: a central player in ARDS pathogenesis Ferroptosis is a regulated, iron-dependent form of cell death distinguished by oxidative damage and lipid peroxidation

Rapid injection can cause localized discomfort and irregular absorption

CRP, an active regulator of host innate immunity, is a biomarker of chronic inflammatory conditions and severe COVID-19 disease, including lung and atherosclerotic disease progression

Cysteine is mainly derived from the trans-sulfuration pathway of methionine and the reduction of cysteine [21]