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The lateral parabrachial nucleus may be a critical site of action as pharmacological activation of GLP-Rs in the lateral parabrachial nucleus (LPBN) inhibits feeding whereas antagonism of LPBN GLP-1Rs induces hyperphagia (Alhadeff et al., The hypothalamus has also been implicated as a major center for mediating the pharmacological effects of GLP-1R action on energy metabolism
Once this bond is broken, the resulting fragments no longer bind GHS-R1a receptors
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